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Although the exact mechanism is not currently known, it is evident that there is a cross-talk between the IGF-1 system, ER and the cognate ER ligand 17-estradiol (E2)

In this study, we subjected HepG2 cells to acute high doses of APAP and compared cell viability, the changes in the intra- and extra-cellular metabolome, as well as carbon flux to that of unexposed controls

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