glutathione brain inflammation Increasing levels by a novel posttranslational mechanism inhibits neuronal hyperexcitability l-glutathione brain inflammation nih Regulation

Sulforaphane metabolites sulforaphane-glutathione, sulforaphane-cysteine-glycine, sulforaphane-cysteine, and sulforaphane N-acetylcysteine (Figure 3) collectively known as dithiocarbamates, are ultimately excreted in the urine (5)

These findings suggest that glutathione amino acid precursors protective mechanism extends to shielding the skin from UV-induced DNA damage, a critical factor in preventing photoaging and skin cancer

RHONDA PATRICK: Well, it doesnt matter either way

In animal models it repaired cognitive deficits at extraordinarily low doses but no human trials of any kind have been published, and its long-term safety is entirely unknown