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Another mechanism involves flavoproteins whose stability relies on FAD mediated folding, such as AIFM1

A recent in vitro study showed that PINK1-dependent phosphorylation of Ser111 of Rab8A antagonistically regulates the phosphorylation of Thr72 of Rab8A by LRRK2 (Vieweg et al., 2020), suggesting that there might be even closer crosstalk between the two master kinase

Supplementation with nutrients from the other micronutrients category (carnitine, choline and taurine) did reverse the ES-induced behavioral deficits (100%)