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The implications for conditions involving compromised blood flow are significant, though human confirmation of this mechanism remains needed

It receives hydride from metabolic processes like glycolysis, the TCA cycle, and fatty acid oxidation to create NADH

This idea is supported by work that shows some genes that drive cancer growth turn on other genes that make intrinsic antioxidants

Simulation of metabolism of GSH in the context of a complete metabolic network of cancer cells and tissues as well as metabolic interactions between cancer cells and their microenvironment is possible with either Boolean models or much more informative genome scale flux models [173]