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The increased susceptibility to cell death could exaggerate inflammation via the release of damage signals (e.g., ATP, High mobility group box 1 protein (HMGB1)) or alternatively, ferroptosis of CD16 + monocytes and/or macrophages that are reputedly more pro-inflammatory than intestinal macrophages could serve to limit inflammation

X-ray diffraction (XRD) analysis revealed a broad and diffuse diffraction pattern without distinct crystalline peaks corresponding to Zn-based phases, indicating an amorphous structure of the EGCGZn nanocomplex (Fig