DISCUSSION BPC 157 was suggested to be a link between inflammatory bowel disease and multiple sclerosis, that could be also used in practice per-orally (as an anti-ulcer peptide, stable in human gastric juice) (5-7, 9), and thereby, being equally effective against the models of both of those diseases
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Proposed mechanisms include upregulation of growth-hormone-receptor expression in injured tissue, VEGF-driven angiogenesis, modulation of the nitric-oxide pathway in vascular endothelium, and activation of FAK-paxillin signaling